These final results indicated that {increased Kliqqi
1
These benefits indicated that greater expression in the Ex4a(+)WT1 isoform may well contribute into the induction of apoptosis. In addition, to look at the effects in the endogenous Ex4a(+)WT1 isoform on Dox-induced apoptosis, K562 cells were being transfected with either of two different WT1 Ex4a-specific siRNAs (si-4a-1 and si-4a-2) or control siRNA (si-control) for twenty-four h, taken care of